Recent Publications

Delmolino LA, Stearns NA, and Castellot JJ. 2001. COP-1, a member of the CCN family, is a heparin-induced growth-arrest specific gene in vascular smooth muscle cells. J. Cell. Physiol. 188:45-55.

Mishra-Gorur K, Singer HA, and Castellot JJ. 2002. The S18 ribosomal protein is a putative substrate for Ca2+/calmodulin-activated protein kinase II. J. Biol. Chem. 277:33537-33540.
Lake AC, Bialik A, Walsh K, and Castellot JJ. 2003. CCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motility. Amer. J. Pathol. 162:219-231.

Mason HR, Nowak RA, Morton CC, and Castellot JJ. 2003. Heparin inhibits the motility and proliferation of human myometrial and leiomyoma smooth muscle cells. Amer J Pathol, 162:1895-1904.

Mason HR, Grove-Strawser D, Rubin BS, Nowak RA, and Castellot JJ. 2004 (in press). Estrogen induces CCN5 expression in the rat uterus in vivo. Endocrinology (published on-line Nov. 6, 2003)

Mason HR, Lake AC, Wubben JE, Nowak RA, and Castellot JJ. 2004 (in press). The growth arrest-specific gene CCN5 is deficient in human leiomyomas and inhibits the proliferation and motility of cultured human uterine smooth muscle cells. Mol. Hum. Reprod.

Lake AC and Castellot JJ. 2004 (in press). CCN5 modulates the antiproliferative effect of heparin and regulates cell motility in vascular smooth muscle cells. Cell Comm. and Signaling., 1:5. (published on-line Nov. 24, 2003)